Classes: Difference between revisions
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This page is a reference taxonomy of pharmacological '''classes''' used throughout Pharmacopedia. Click any class name to browse its members. Individual | This page is a reference taxonomy of pharmacological '''classes''' used throughout Pharmacopedia. Click any class name to browse its members. Individual meds are listed on the [[List of CNS-active medicines]] page. | ||
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=== Monoamine Oxidase Inhibitors (MAOIs) === | === Monoamine Oxidase Inhibitors (MAOIs) === | ||
Inhibit MAO-A and/or MAO-B, increasing monoamine levels. Require strict dietary and | Inhibit MAO-A and/or MAO-B, increasing monoamine levels. Require strict dietary and med interaction precautions. Subclasses: | ||
* '''Irreversible non-selective MAOIs''' — inhibit both MAO-A and MAO-B permanently | * '''Irreversible non-selective MAOIs''' — inhibit both MAO-A and MAO-B permanently | ||
* '''Reversible MAO-A inhibitors (RIMAs)''' — safer interaction profile; displaceable by tyramine | * '''Reversible MAO-A inhibitors (RIMAs)''' — safer interaction profile; displaceable by tyramine | ||
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=== Beta Blockers (peripheral somatic anxiolytic) === | === Beta Blockers (peripheral somatic anxiolytic) === | ||
β-adrenergic receptor antagonists. While primarily cardiovascular | β-adrenergic receptor antagonists. While primarily cardiovascular meds, they are widely used off-label as peripheral anxiolytics — blunting the somatic (tachycardia, tremor, sweating) manifestations of acute anxiety without sedation or dependence liability. Particularly useful for performance anxiety, akathisia, and essential tremor. [[Propranolol]] is the prototype. See [[:Category:Beta Blockers]]. | ||
== Stimulants & Wake-Promoting Agents == | == Stimulants & Wake-Promoting Agents == | ||
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Many anticonvulsants are also used for pain, migraine prophylaxis, mood stabilization, or anxiety. | Many anticonvulsants are also used for pain, migraine prophylaxis, mood stabilization, or anxiety. | ||
== Antiparkinsonian | == Antiparkinsonian Meds == | ||
=== Dopamine Precursors === | === Dopamine Precursors === | ||
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Reduce glutamate-mediated excitotoxicity and may reduce dyskinesias. Amantadine also has dopamine-releasing effects. | Reduce glutamate-mediated excitotoxicity and may reduce dyskinesias. Amantadine also has dopamine-releasing effects. | ||
== Anti-Dementia | == Anti-Dementia Meds == | ||
=== Acetylcholinesterase Inhibitors (AChEIs) === | === Acetylcholinesterase Inhibitors (AChEIs) === | ||
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* '''Second-generation''' — less CNS penetration; non-sedating | * '''Second-generation''' — less CNS penetration; non-sedating | ||
== Addiction | == Addiction Med == | ||
Meds used to treat substance use disorders. Mechanisms are specific to the target substance: | |||
* Opioid agonist therapy (methadone, buprenorphine) | * Opioid agonist therapy (methadone, buprenorphine) | ||
* Opioid antagonists (naltrexone, naloxone, nalmefene) | * Opioid antagonists (naltrexone, naloxone, nalmefene) | ||
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* Alpha-2 agonists for withdrawal (clonidine, lofexidine) | * Alpha-2 agonists for withdrawal (clonidine, lofexidine) | ||
== Migraine | == Migraine Meds == | ||
=== Triptans (5-HT1B/1D Agonists) === | === Triptans (5-HT1B/1D Agonists) === | ||
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== Research Materials == | == Research Materials == | ||
Compounds primarily of research interest, with limited or no human clinical approval. Many are analogues of approved | Compounds primarily of research interest, with limited or no human clinical approval. Many are analogues of approved meds or classical psychoactive substances. Use of the term '''material''' is preferred over "med" or "substance" in this context. | ||
=== Lysergamides === | === Lysergamides === | ||