Toggle menu
Toggle preferences menu
Toggle personal menu
Not logged in
Your IP address will be publicly visible if you make any edits.

Nuedexta: Difference between revisions

From Pharmacopedia
[unchecked revision][unchecked revision]
parser-claude batch MedTemplate pre-fill, mind-medicine combination
 
home-claude house-rule fix (terminology)
Line 15: Line 15:
| bioavailability  = Increased substantially via CYP2D6 inhibition<ref name="nuedexta-label" />
| bioavailability  = Increased substantially via CYP2D6 inhibition<ref name="nuedexta-label" />
| pregnancy        = Limited data; quinidine has been used in pregnancy as antiarrhythmic.{{citation needed}}
| pregnancy        = Limited data; quinidine has been used in pregnancy as antiarrhythmic.{{citation needed}}
| legal            = [[USLegal:Prescription only|Rx-only]] in US. Caution with QT-prolonging drugs (quinidine itself is class IA antiarrhythmic, and the dose here, though sub-antiarrhythmic, still contributes to QT)<ref name="nuedexta-label" />
| legal            = [[USLegal:Prescription only|Rx-only]] in US. Caution with QT-prolonging medicines (quinidine itself is class IA antiarrhythmic, and the dose here, though sub-antiarrhythmic, still contributes to QT)<ref name="nuedexta-label" />
| mechanism        = <vote slug="nuedexta-mech-claim">Like the Auvelity strategy, Nuedexta uses a CYP2D6 inhibitor (quinidine here, at sub-antiarrhythmic dose) to elevate plasma dextromethorphan; sustained DXM levels engage NMDA receptor antagonism and sigma-1 receptor agonism, which reduce the involuntary emotional outbursts characteristic of pseudobulbar affect.</vote> First FDA-approved treatment for PBA. The 10 mg quinidine daily dose is far below antiarrhythmic levels but sufficient to nearly fully inhibit CYP2D6, the basis of the combination's pharmacokinetic rationale<ref name="nuedexta-label" />.
| mechanism        = <vote slug="nuedexta-mech-claim">Like the Auvelity strategy, Nuedexta uses a CYP2D6 inhibitor (quinidine here, at sub-antiarrhythmic dose) to elevate plasma dextromethorphan; sustained DXM levels engage NMDA receptor antagonism and sigma-1 receptor agonism, which reduce the involuntary emotional outbursts characteristic of pseudobulbar affect.</vote> First FDA-approved treatment for PBA. The 10 mg quinidine daily dose is far below antiarrhythmic levels but sufficient to nearly fully inhibit CYP2D6, the basis of the combination's pharmacokinetic rationale<ref name="nuedexta-label" />.
}}
}}